Department of Pharmacology
Chiba University, Graduate School of Medicine

CONTENTS
TOP PAGE
RESEARCH THEMES
MEMBERS
PUBLICATIONS
ACCESS
LINK
Japanese Page
Publications(2005)
1)Saito T, Sato T, Miki T, Seino S, Nakaya H.
Role of ATP-sensitive K+ channels in electrophysiological alterations during myocardial ischemia: a study using Kir6.2-null mice.
Am J Physiol Heart Circ Physiol 2005 Jan; 288(1): H352-7.

2)Sato T, Saito T, Saegusa N, Nakaya H.
Mitochondrial Ca2+-activated K+ channels in cardiac myocytes: a mechanism of the cardioprotective effect and modulation by protein kinase A.
Circulation 2005 Jan; 111(2): 198-203.

3)Harada M, Qin Y, Takano H, Minamino T, Zou Y, Toko H, Ohtsuka M, Matsuura K, Sano M, Nishi J, Iwanaga K, Akazawa H, Kunieda T, Zhu W, Hasegawa H, Kunisada K, Nagai T, Nakaya H, Yamauchi-Takihara K, Komuro I.
G-CSF prevents cardiac remodeling after myocardial infarction by activating the Jak-Stat pathway in cardiomyocytes.
Nat Med 2005 Mar; 11(3): 305-11.

4)Shinmura K, Tamaki K, Sato T, Ishida H, Bolli R.
Prostacyclin attenuates oxidative damage of myocytes by opening mitochondrial ATP-sensitive K+ channels via the EP3 receptor.
Am J Physiol Heart Circ Physiol 2005 May; 288(5): H2093-101.

5)Saegusa N, Sato T, Saito T, Tamagawa M, Komuro I, Nakaya H.
Kir6.2-deficient mice are susceptible to stimulated ANP secretion: KATP channel acts as a negative feedback mechanism?
Cardiovasc Res 2005 Jul; 67(1): 60-8.

6)Bai CX, Kurokawa J, Tamagawa M, Nakaya H, Furukawa T.
Nontranscriptional regulation of cardiac repolarization currents by testosterone.
Circulation 2005 Sep; 112(12): 1701-10.
  • Publications Top